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Research Grade : For laboratory research only

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Broad or systemic tissue repair research: TB-500's actin-mediated, migration-promoting mechanism is associated with wound closure and tissue-repair research that is more distributed across the body, including cardiac and corneal models

As MOTS-c activates AMPK and NRF2, it also indirectly suppresses the mTOR (mechanistic target of rapamycin) pathway, the master growth and anabolism regulator that, when chronically overactivated, accelerates the accumulation of senescent cells

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Current Opinion in Investigational Drugs, 7(4), 312-318