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In addition to downregulation of injury and inflammation-related pathways, GSEA also revealed suppression of adipogenesis-related pathways in the liver of GPR146-deficient mice, suggesting that GPR146 may promote hepatic steatosis via PPAR-mediated transcriptional programs (Fig

MASH and ALD share several similarities, including excess intake of nutrients or ethanol, and the pathological features of hepatic steatosis, inflammation and fibrosis 5,6

Deactivation of SpyLock reactivity within an assay To explore whether SpyLock reactivity could be turned off directly during an assay, we performed ELISAs with directed immobilization via SpyLock

& Garfield, A

Int J Mol Sci 20(9):2269